Did you get ME/CFS after a vaccine?

Did you get sick after a vaccine? Then maybe you should consider an immune illness due to intoxication to aluminium.

It is called Macrophagic Myofasciitis and often confused with Chronique Fatigue Syndrome.
The symptoms are muscle pain (95%), fatigue, post-exertional malaise, joint pain (50-60%) and febrile syndrome (30%). It can only be diagnosed through muscle biopsy.

Most manufacturers of vaccines add aluminium to vaccines to enhance their action. The WHO already warned the doses are however far too high and the information leaflets too vague about the composition.
Many doctors have no clue about the toxic doses of vaccines.
Dr Jean Pilette studied closely the composition and calculated that babies get up to 170 times the allowed toxic doses in his blood serum several times a year.

If you got a few vaccines, subcutaneous nodules can form. Micro-analysis has shown the presence of aluminium in the macrophages of these nodules.


What else contains a lot of aluminium?
- gastric reflux medication
- baby milk (especially based on soya)
- tap water (it is used to clean dirty water)
- pesticides
- vaginal showers
- black tea
- melted cheese products
- food additives E173, E521, E522, E523, E554, E555, E556, E559
- aluminium frying pan
- percolator
- deodorant
- industrial work

Aluminium is an iron widely present in nature and often used by man. It is however very toxic for the nervous system because it passes easily through the brain blood barrier. Then it can access delicate parts of the deep brain.
Aluminium has also a negative influence on the Krebs cycle. This cycle is essential in the wellbeing and health of every individual. When the Krebs cycle is interrupted, the human system goes down. The engine of your cells brake down, they are poisoned.

Macrophagic Myofasciitis is considered a rare disease. Chances your doctor has never heard about it.

Sources:
http://www.orpha.net/data/patho/GB/uk-myofa.pdf
http://www.ncbi.nlm.nih.gov/pubmed/12797472
http://brain.oxfordjournals.org/content/124/5/974.full

http://onlinelibrary.wiley.com/doi/10.1046/j.1432-1033.2000.01328.x/full
The most important function of mitochondria is the oxidation of substrates to produce bioenergy. Several lines of evidence indicate that mitochondria from neurons affected by degenerative disorders related to aging exhibit aberrant oxidation processes [16]. Furthermore, it is worth mentioning that there is a strict correlation between Krebs cycle and glycolysis. In this connection it has been demonstrated that Al is a strong inhibitor of some enzymes of the glycolysis pathway [17–19] as well as glucokinase and phosphofructokinase [20]. All these elements reinforce the possibility that Al could interfere the bioenergetic pathways in mitochondria.
...
In addition, it has been demonstrated that aluminum alters the homeostasis of intracellular calcium in mitochondria, a phenomenon linked to initiation of apoptotic cell death; this observation provides support for the potential detrimental effects of this metal ion on this organelle [53].

Agammaglobulinemia makes you susceptible to chronic enteroviral infections of CNS

http://www.ncbi.nlm.nih.gov/pubmed/3296100?dopt=Abstract

Chronic enteroviral meningoencephalitis in agammaglobulinemic patients.


McKinney RE Jr, Katz SL, Wilfert CM.
Rev Infect Dis. 1987 Mar-Apr;9(2):334-56.


Abstract

Patients with agammaglobulinemia are particularly susceptible to chronic enteroviral infections of the central nervous system. Data on 42 patients were obtained by literature review, communications with other physicians, and personal experiences. Thirty-eight patients had congenital immunodeficiencies, most frequently X-linked agammaglobulinemia. Most patients who could be assessed were receiving maintenance therapy with intramuscular gamma-globulin before their enteroviral infection. Seven patients had not been recognized as hypogammaglobulinemic before the onset of infection. The commonest pathogens were echoviruses (37 of 41 cases), especially type 11 (11 cases). Thus far, four patients have had sequential or simultaneous infections with a second enteroviral serotype. Other features of the disease have included weakness, lethargy or coma, headaches, hearing loss, seizures, ataxia, and paresthesias. Some patients have also had nonneurologic manifestations of chronic enteroviral infection, including fever, the dermatomyositis-like syndrome, edema, rashes, and hepatitis. Treatment has consisted primarily of antibody administration, either in intravenous immunoglobulin preparations or in immune plasma. Twelve patients have received intraventricular immunoglobulin through reservoir devices; six of these 12 have improved substantially, as judged by clinical criteria.

PMID: 3296100 [PubMed - indexed for MEDLINE]

Sources:

http://www.ncbi.nlm.nih.gov/pubmed/3296100?dopt=Abstract
http://www.jacionline.org/article/S0091-6749(01)70038-3/abstract