Could it be a coincidence I had tuberculosis as a child and then developed lupus as a teenager, ME as years went by and now arthritis?
What if dysfunctional methylation leads to disease?
http://www.ncbi.nlm.nih.gov/pubmed/21168462
Hum Immunol. 2011 Mar;72(3):262-8. Epub 2010 Dec 16.
Vitamin D receptor gene methylation is associated with ethnicity, tuberculosis, and TaqI polymorphism.
Andraos C, Koorsen G, Knight JC, Bornman L.
Department of Biochemistry, University of Johannesburg, Auckland Park, South Africa.
Abstract
The Vitamin D receptor (VDR) gene encodes a transcription factor which, on activation by vitamin D, modulates diverse biologic processes, including calcium homeostasis and immune function. Genetic variation involving VDR shows striking differences in allele frequency between populations and has been associated with disease susceptibility, including tuberculosis and autoimmunity, although results have often been conflicting.
We hypothesized that methylation of VDR may be population specific and that the combination of differential methylation and genetic variation may characterize tuberculosis (TB) predisposition. We use bisulfite conversion and/or pyrosequencing to analyze the methylation status of 17 CpGs of VDR and to genotype 7 SNPs in the 3' CpG Island (CpG island [CGI] 1060), including the commonly studied SNPs ApaI (rs7975232) and TaqI (rs731236).
We show that, for lymphoblastoid cell lines from two ethnically diverse populations (Yoruba from HapMap, n = 30 and Caucasians, n = 30) together with TB cases (n = 32) and controls (n = 29) from the Venda population of South Africa, there are methylation variable positions in the 3' end that significantly distinguish ethnicity (9/17 CpGs) and TB status (3/17 CpGs).
Moreover, methylation status shows complex association with TaqI genotype highlighting the need to consider both genetic and epigenetic variants in genetic studies of VDR association with disease.
Copyright © 2011 American Society for Histocompatibility and Immunogenetics. Published by Elsevier Inc. All rights reserved.
Source: http://www.ncbi.nlm.nih.gov/pubmed/21168462
donderdag 8 maart 2012
zondag 4 maart 2012
zaterdag 3 maart 2012
Cholesterol drug can be responsible for permanent impairment of memory
It seems I'm not the only one who suffered total black-outs from this drug.
www.spacedoc.net/lipitor.htm
All statins in use today can be associated with cognitive side effects such as amnesia, forgetfulness, confusion and disorientation. The mechanism of action seems to be excessive reduction in the bio-availability of cholesterol for proper brain function.
For some reason Lipitor "does this better" than the others just as Baycol caused rhabdomyolysis so much more than the other statins that it had to be removed from the market. But, in truth, all the statins can cause this dreaded muscle breakdown.
Associated with this distortion of memory is a tendency for prolonged, even permanent impairment of the process of short-term memory with the use of Lipitor. We have a growing list of these reports where former breadwinners have been reduced to dependents and the entire family structure has been drastically altered.
The following are but a few examples of this legacy of Lipitor - atorvastatin - sent to me by readers.
1) Three weeks ago I had an eight-hour episode of TGA. I had been on Lipitor and Lisinopril for about six weeks prior. I have stopped both medications for the time being until I get back to normal. Even after stopping the Lipitor I was disoriented (especially in the morning) and "just wasn't feeling right (poorly described by a physician).
2) Four months ago I was put on Lipitor to reduce my bad cholesterol to 100. Suddenly I found I could not handle basic math or remember how to spell. It became so bad that I was in a constant fog. I should tell you I spent most of my career in Silicon Valley writing specifications for software and hold a patent on expert system technology. I had an MRI to rule out a brain tumor or stroke. Since the only thing that had changed was the addition of Lipitor I stopped taking it. Five weeks later I am still having problems spelling and frequently forget things.
3) We have recently taken my mother off of the Statins because she is suffering from memory loss. Ten days after removing her from Statins, her doctor sent her for neuropsych testing and a CT scan. CT results are still pending. The neuropsychologist diagnosed Alzheimer's disease and referred her back to her internist to begin cholinesterase inhibitors. We believe that this is a premature diagnosis, as her memory loss has not progressed beyond the initial stages observed over 2.5 years ago and that she has only been off Statins for ten days when she was tested. She has been on statin therapy for nine years.
4) I was put on Lipitor when Mevacor didn't appear to be doing its job. I was started on Mevacor at approximately 30 years of age....I'm 49 now. The Lipitor was started approximately two years ago...I'd have to have the Doctor's office check as I can't really remember when I was switched! I noticed my memory was getting bad and every doctor I saw blamed it on menopause and / or my fibromyalgia.
Within the past few months however, new and frightening symptoms appeared. While talking to friends, out shopping or just sitting home and watching television I would have the strangest and most unsettling sensation that I didn't know who I was. It would last for seconds and felt so odd- like an out of body experience...who was this person talking...oh, it must be me.
I was always a voracious reader and now it's so hard for me to concentrate and remember things that I've all but given up reading. Crossword puzzles were a favorite pastime. Now I have a hard time carrying on a conversation since I stutter and ummm and uhhh trying to think of a word or name. Names of old friends escape me and sometimes I can't recall if I've done something just seconds after I've done it.
The muscles in my forearms began to be so sore I found it difficult to take a half-gallon container of soymilk out of the fridge, and pushing the button on the remote to change channels actually hurt. I was terrified that I had Alzheimer's, or worse yet a brain tumor. I was prepared to ask my Doctor for a CT scan or MRI to rule out that possibility when I received the e-newsletter and discovered the root of my problem. I immediately discontinued my Lipitor.
When I showed my physician the information I had gathered about Statins she was very interested as her own husband is on 20mg of Lipitor and had been complaining of memory problems and brain fog! She said she had no idea the two were linked.
...
www.spacedoc.net/lipitor.htm
www.spacedoc.net/lipitor.htm
All statins in use today can be associated with cognitive side effects such as amnesia, forgetfulness, confusion and disorientation. The mechanism of action seems to be excessive reduction in the bio-availability of cholesterol for proper brain function.
For some reason Lipitor "does this better" than the others just as Baycol caused rhabdomyolysis so much more than the other statins that it had to be removed from the market. But, in truth, all the statins can cause this dreaded muscle breakdown.
Associated with this distortion of memory is a tendency for prolonged, even permanent impairment of the process of short-term memory with the use of Lipitor. We have a growing list of these reports where former breadwinners have been reduced to dependents and the entire family structure has been drastically altered.
The following are but a few examples of this legacy of Lipitor - atorvastatin - sent to me by readers.
1) Three weeks ago I had an eight-hour episode of TGA. I had been on Lipitor and Lisinopril for about six weeks prior. I have stopped both medications for the time being until I get back to normal. Even after stopping the Lipitor I was disoriented (especially in the morning) and "just wasn't feeling right (poorly described by a physician).
2) Four months ago I was put on Lipitor to reduce my bad cholesterol to 100. Suddenly I found I could not handle basic math or remember how to spell. It became so bad that I was in a constant fog. I should tell you I spent most of my career in Silicon Valley writing specifications for software and hold a patent on expert system technology. I had an MRI to rule out a brain tumor or stroke. Since the only thing that had changed was the addition of Lipitor I stopped taking it. Five weeks later I am still having problems spelling and frequently forget things.
3) We have recently taken my mother off of the Statins because she is suffering from memory loss. Ten days after removing her from Statins, her doctor sent her for neuropsych testing and a CT scan. CT results are still pending. The neuropsychologist diagnosed Alzheimer's disease and referred her back to her internist to begin cholinesterase inhibitors. We believe that this is a premature diagnosis, as her memory loss has not progressed beyond the initial stages observed over 2.5 years ago and that she has only been off Statins for ten days when she was tested. She has been on statin therapy for nine years.
4) I was put on Lipitor when Mevacor didn't appear to be doing its job. I was started on Mevacor at approximately 30 years of age....I'm 49 now. The Lipitor was started approximately two years ago...I'd have to have the Doctor's office check as I can't really remember when I was switched! I noticed my memory was getting bad and every doctor I saw blamed it on menopause and / or my fibromyalgia.
Within the past few months however, new and frightening symptoms appeared. While talking to friends, out shopping or just sitting home and watching television I would have the strangest and most unsettling sensation that I didn't know who I was. It would last for seconds and felt so odd- like an out of body experience...who was this person talking...oh, it must be me.
I was always a voracious reader and now it's so hard for me to concentrate and remember things that I've all but given up reading. Crossword puzzles were a favorite pastime. Now I have a hard time carrying on a conversation since I stutter and ummm and uhhh trying to think of a word or name. Names of old friends escape me and sometimes I can't recall if I've done something just seconds after I've done it.
The muscles in my forearms began to be so sore I found it difficult to take a half-gallon container of soymilk out of the fridge, and pushing the button on the remote to change channels actually hurt. I was terrified that I had Alzheimer's, or worse yet a brain tumor. I was prepared to ask my Doctor for a CT scan or MRI to rule out that possibility when I received the e-newsletter and discovered the root of my problem. I immediately discontinued my Lipitor.
When I showed my physician the information I had gathered about Statins she was very interested as her own husband is on 20mg of Lipitor and had been complaining of memory problems and brain fog! She said she had no idea the two were linked.
...
www.spacedoc.net/lipitor.htm
vrijdag 2 maart 2012
woensdag 29 februari 2012
Survey: The statin study effect
https://www.statineffects.com/
Have you had any side effects from statin treatment?
If you would like to share your experience on statins with researchers at the University of California in San Diego ,
please email at statinstudy@ucsd.edu
and/or complete an online (paperless) survey at
http://www.statineffects.com
Have you had any side effects from statin treatment?
If you would like to share your experience on statins with researchers at the University of California in San Diego ,
please email at statinstudy@ucsd.edu
and/or complete an online (paperless) survey at
http://www.statineffects.com
zondag 26 februari 2012
The hidden cure for viral diseases?
http://www.ceri.com/BHTbook-StevenWmFowkes-100903.pdf
This online book offers a biologically sustainable solution to chronic viral disease (herpes, CMV, ...)
The treatment is about the drug BTH (butylated hydroxytoluene). It will cost you 10$ in bulk or 50$ in capsules. Show it to your doctor.
From what I read, it looks promising. Take your time or have someone take notes for you.
This online book offers a biologically sustainable solution to chronic viral disease (herpes, CMV, ...)
The treatment is about the drug BTH (butylated hydroxytoluene). It will cost you 10$ in bulk or 50$ in capsules. Show it to your doctor.
From what I read, it looks promising. Take your time or have someone take notes for you.
vrijdag 24 februari 2012
Duidelijkheid rond vitamine B12
Naar aanleiding van de ervaringen van een patient met vitamine B12, deel ik hier zijn vaststellingen.
Hij was ernstig beperkt gedurende vele jaren maar is nu bijna volledig hersteld. De moeite waard om het eens door te nemen. Veel mensen prikken of slikken immers vitamine B12.
http://aboutmecfs.org.violet.arvixe.com/Trt/B12Ineffectiveness.aspx
Zelf heb ik geëxperimenteerd met verscheidene injecteerbare producten omdat de vloeistof zo pijnlijk blijkt. Soms kon ik gedurende een dag mijn bed niet meer uit van de pijn. Andere mensen hebben hier ook last van, anderen dan weer niet.
Mogelijk ligt het antwoord in zijn (vertaalde) tekst hieronder.
Niet iedereen blijkt "vitamine B12" te kunnen omzetten. De oorzaken zijn divers:
Hij was ernstig beperkt gedurende vele jaren maar is nu bijna volledig hersteld. De moeite waard om het eens door te nemen. Veel mensen prikken of slikken immers vitamine B12.
Hier vindt u het origineel:
http://aboutmecfs.org.violet.arvixe.com/Trt/B12Ineffectiveness.aspx
Zelf heb ik geëxperimenteerd met verscheidene injecteerbare producten omdat de vloeistof zo pijnlijk blijkt. Soms kon ik gedurende een dag mijn bed niet meer uit van de pijn. Andere mensen hebben hier ook last van, anderen dan weer niet.
Mogelijk ligt het antwoord in zijn (vertaalde) tekst hieronder.
Niet iedereen blijkt "vitamine B12" te kunnen omzetten. De oorzaken zijn divers:
- Onmogelijkheid om vit B12 te absorberen vanwege pernicieuze anemie (genetisch of auto-immuun)
- Men kan vit B12 niet binden voor transport
- Er zijn genetische uitdagingen
- Verlaagde cobalamine niveaus in ruggemerg - hypothetische oorzaak, bevestigd probleem
- Afwezigheid van enzym om methylb12 in adenosylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om adenosylb12 in methylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om cyanocobalamin in methylb12 of adenosylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om hydroxycobalamin in methylb12 of adenosylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om glutathionylcobalamin in methylb12 of adenosylb12 om te zetten - hypothetisch, enige aanwijzingen voor probleem
- Te weinig enzymen om foliumzuur om te zetten in de bio-actieve vorm, methylfolaat - bevestigde oorzaak
- Actieve vorm: vit B12 in orale vorm wordt slechts voor 1% opgenomen omdat het door een klein celgaatje moet en omdat de binding verminderd kan zijn door genetische of aangeworven redenen.
- Inactieve vorm = cyanoB12 of hydroxyB12
- voordeel: waarden in bloed stijgen snel boven 300 pg/ml
- nadeel: geneest de schade niet aangericht door tekort aan vit B12
- nadeel: mogelijk blokkeren ze de B12 receptoren op de cellen waardoor de echte B12 niet meer kan werken
- nadeel: beide vormen kunnen maar voor 10 mcg/dag door een celgaatje en dus beperkt omgezet worden
- nadeel: ze doen het niveau van de ongebonden cobalamine niet stijgen (en net deze heb je nodig voor herstel)
- Enzymen: Sommige mensen missen de enzymen om cyanoB12 en hydroxyB12 om te zetten in de actieve vorm
- Verkeerd gebruik: Zuigtabletten blijken alleen te werken als je ze gedurende 45 min tot 2 uur onder de bovenlip of onder de tong laat smelten.
- Verkeerd merk: Slechts enkele merken blijken effectief werkzaam waaronder:
- Enzymatic Therapy 1 mg
- Jarrow Formulas 1 en 5 mg
- Country Life Dibenzodide 1 mg (adenosylB12)
- Solgar Metalfolin (methylfolaat) is beter dan eender welk foliumzuur
- Injecteerbaar product: MethylB12 wordt afgebroken tot hydroxyB12 maar geneest geen mensen met een tekort aan cobalamine. Dat vereist ongebonden methylB12 én adenosylB12. Mensen nemen vaak slechts 1 soort vitamine B12.
- Te lage dosering: mensen nemen te lage doseringen om efficient het ruggemerg te beïnvloeden
- Te weinig methylfolaat: dit is de werkzame vorm van foliumzuur
- Tekort aan essentiële co-factoren
- Suppleren met glutathion of precursors van glutathion: kunnen mogelijk een actieve vit B12 deficiëntie inleiden
Persoonlijk heb ik een vraagteken bij het punt dat het geen schade zou herstellen n.a.v. een tekort. Zelfs indien het geen schade herstelt, merkte ik het eerste jaar toch een mindering van de brainfog en meer stamina zo'n 24 uur na een inspuiting. Hoe meer tijd ik tussen de spuitjes liet, hoe pijnlijker ze telkens weer werden.
Voor degenen die er meer over willen lezen:
Het verhaal van Fred: http://aboutmecfs.org/Trt/B12Story.aspx
Een intro over B12: http://aboutmecfs.org/Trt/B12Intro.aspx
Het behandelingsplan van Fred: http://aboutmecfs.org/Trt/B12Treatment.aspx
http://www.iherb.com/Jarrow-Formulas-Methyl-B-12-5000-mcg-60-Lozenges/117?at=0
http://www.iherb.com/Country-Life-Gluten-Free-Active-B-12-Dibencozide-3000-mcg-60-Lozenges/1637?at=0
http://www.iherb.com/Solgar-Folate-Metafolin-Folic-Acid-800-mcg-100-Tablets/13961?at=0
http://www.iherb.com/Natural-Factors-Potassium-Citrate-99-mg-90-Tablets/2627?at=0
EXTRA LINK: http://www.diagned.nl/files_content/Diagned38-3.pdf
Een intro over B12: http://aboutmecfs.org/Trt/B12Intro.aspx
Het behandelingsplan van Fred: http://aboutmecfs.org/Trt/B12Treatment.aspx
http://www.iherb.com/Jarrow-Formulas-Methyl-B-12-5000-mcg-60-Lozenges/117?at=0
http://www.iherb.com/Country-Life-Gluten-Free-Active-B-12-Dibencozide-3000-mcg-60-Lozenges/1637?at=0
http://www.iherb.com/Solgar-Folate-Metafolin-Folic-Acid-800-mcg-100-Tablets/13961?at=0
http://www.iherb.com/Natural-Factors-Potassium-Citrate-99-mg-90-Tablets/2627?at=0
EXTRA LINK: http://www.diagned.nl/files_content/Diagned38-3.pdf
maandag 20 februari 2012
Statin Adverse Effects: Evidence for a Mitochondrial Mechanism
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2849981/?tool=pmcentrez
Statin Adverse Effects: A Review of the Literature and Evidence for a Mitochondrial Mechanism
In the case of statins, a potential basis for opposing effects occurring in muscle and in other organs can be identified. Evidence supports the proposition that antioxidant effects of statins underlie (or contribute to) many fundamental statin benefits – including benefits to flow and oxygen delivery46-48 and inflammation.49, 50 These effects may participate in improved walking distance in patients on statins, including benefits to muscle/walking in persons with and without peripheral artery disease.29 Yet a subset of people reproducibly exhibit increases in markers of oxidation on statins,51 and the occurrence of this increase has been tied to muscle pain on statins.52, 53
...
A range of cases have now been reported in which statin use has “uncovered” previously clinically silent or clinically tolerated conditions, ranging from McArdle disease159, 160 to myotonic dystrophy159 to acid maltase deficiency161 to possible Kennedy disease.159 Statins have also exacerbated known muscle conditions, such as myasthenia gravis.78 In the case of mitochondrial myopathies, the relative degree to which statins have unmasked vs induced disease may not always be clear.159, 162
...
Several widely used statins – atorvastatin, simvastatin, and lovastatin (and previously cerivastatin, now off the market) – are metabolized by the cytochrome P450 (CYP)3A4 pathway.318
...
While a medley of potential mechanisms may cause or contribute to statin AEs (and these merit more full review in another venue), mitochondrial mechanisms have been repeatedly implicated in muscle AEs. Mitochondrial defects predispose to problems on statins (as shown in the second to last entry of Table IV, ‘Genetic mutations associated with mitochondrial dysfunction’). Additionally, statins predispose to mitochondrial defects (Table V,22 31, 32, 112, 155, 158, 162, 397, 406-414) – in all users and, to a greater degree, in vulnerable individuals. Dose-dependent reductions in coenzyme Q1020-22 can reduce cell energy, promote oxidation,362, 415 promote apoptosis, and unmask silent mitochondrial defects.23-25, 362, 415-418 The mevalonate pathway, which statins inhibit, also produces heme-A, which has it own central involvement in mitochondrial electron transport.419
...
For instance, mitochondrial encephalomyopathy resulting from heritable coenzyme Q10 deficiency classically produces fatigue, muscle symptoms, and cognitive problems,440 although the cases referred for analysis are often relatively severe.429, 441 Gastrointestinal26 and neurological symptoms,432, 442 psychiatric symptoms,443-446 sleep problems,444, 447 glucose elevations,182 and a range of other symptoms reported on statins also arise in mitochondrial dysfunction.379, 448-457
...
Thus, in an analysis of data, presented in the Australian Adverse Drug Reaction Bulletin, it was noted that “Statin-associated peripheral neuropathy may persist for months or years after withdrawal of the statin… In two ADRAC (Adverse Drug Reactions Advisory Committee) cases of persistent peripheral neuropathy, motor and sensory conduction tests showed minimal recovery 4 and 12 months, respectively, after discontinuation of simvastatin, despite clinical improvement.”561
As reviewed here, AEs on statins may signal a mitochondrial vulnerability, which may alter or perhaps even reverse an otherwise favorable impact of statins on cell energetics. And AEs may signal occurrence of a net prooxidant rather than antioxidant effect of statins53 with possible unfavorable implications for a range of statins' proposed pleiotropic effects.892
Full report here: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2849981/?tool=pmcentrez
Statin Adverse Effects: A Review of the Literature and Evidence for a Mitochondrial Mechanism
In the case of statins, a potential basis for opposing effects occurring in muscle and in other organs can be identified. Evidence supports the proposition that antioxidant effects of statins underlie (or contribute to) many fundamental statin benefits – including benefits to flow and oxygen delivery46-48 and inflammation.49, 50 These effects may participate in improved walking distance in patients on statins, including benefits to muscle/walking in persons with and without peripheral artery disease.29 Yet a subset of people reproducibly exhibit increases in markers of oxidation on statins,51 and the occurrence of this increase has been tied to muscle pain on statins.52, 53
...
A range of cases have now been reported in which statin use has “uncovered” previously clinically silent or clinically tolerated conditions, ranging from McArdle disease159, 160 to myotonic dystrophy159 to acid maltase deficiency161 to possible Kennedy disease.159 Statins have also exacerbated known muscle conditions, such as myasthenia gravis.78 In the case of mitochondrial myopathies, the relative degree to which statins have unmasked vs induced disease may not always be clear.159, 162
...
Several widely used statins – atorvastatin, simvastatin, and lovastatin (and previously cerivastatin, now off the market) – are metabolized by the cytochrome P450 (CYP)3A4 pathway.318
...
While a medley of potential mechanisms may cause or contribute to statin AEs (and these merit more full review in another venue), mitochondrial mechanisms have been repeatedly implicated in muscle AEs. Mitochondrial defects predispose to problems on statins (as shown in the second to last entry of Table IV, ‘Genetic mutations associated with mitochondrial dysfunction’). Additionally, statins predispose to mitochondrial defects (Table V,22 31, 32, 112, 155, 158, 162, 397, 406-414) – in all users and, to a greater degree, in vulnerable individuals. Dose-dependent reductions in coenzyme Q1020-22 can reduce cell energy, promote oxidation,362, 415 promote apoptosis, and unmask silent mitochondrial defects.23-25, 362, 415-418 The mevalonate pathway, which statins inhibit, also produces heme-A, which has it own central involvement in mitochondrial electron transport.419
...
For instance, mitochondrial encephalomyopathy resulting from heritable coenzyme Q10 deficiency classically produces fatigue, muscle symptoms, and cognitive problems,440 although the cases referred for analysis are often relatively severe.429, 441 Gastrointestinal26 and neurological symptoms,432, 442 psychiatric symptoms,443-446 sleep problems,444, 447 glucose elevations,182 and a range of other symptoms reported on statins also arise in mitochondrial dysfunction.379, 448-457
...
Thus, in an analysis of data, presented in the Australian Adverse Drug Reaction Bulletin, it was noted that “Statin-associated peripheral neuropathy may persist for months or years after withdrawal of the statin… In two ADRAC (Adverse Drug Reactions Advisory Committee) cases of persistent peripheral neuropathy, motor and sensory conduction tests showed minimal recovery 4 and 12 months, respectively, after discontinuation of simvastatin, despite clinical improvement.”561
As reviewed here, AEs on statins may signal a mitochondrial vulnerability, which may alter or perhaps even reverse an otherwise favorable impact of statins on cell energetics. And AEs may signal occurrence of a net prooxidant rather than antioxidant effect of statins53 with possible unfavorable implications for a range of statins' proposed pleiotropic effects.892
Full report here: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2849981/?tool=pmcentrez
Dr. Klinghardt's Treatment of Lyme Disease
http://articles.mercola.com/sites/articles/archive/2009/08/04/Dr-Klinghardts-Treatment-of-Lyme-Disease.aspx
Extract:
Regular lab parameters affected by Lyme:
•Abnormal lipid profile (moderate cholesterol elevation with significant LDL elevation)
•Insulin resistance
•Borderline low white blood cells, normal SED rate and CRP
•Normal thyroid hormone tests but positive Barnes test and excellent response to giving T3
•Type 2 (high cortisol, low DHEA) or type 3 adrenal failure (low cortisol and DHEA)
•Low testosterone and DHEA
•Decreased urine concentration (low specific gravity)
•Complex changes in cytokines, interferones, NK cells, white blood cell indicators, etc.
...
Extract:
Regular lab parameters affected by Lyme:
•Abnormal lipid profile (moderate cholesterol elevation with significant LDL elevation)
•Insulin resistance
•Borderline low white blood cells, normal SED rate and CRP
•Normal thyroid hormone tests but positive Barnes test and excellent response to giving T3
•Type 2 (high cortisol, low DHEA) or type 3 adrenal failure (low cortisol and DHEA)
•Low testosterone and DHEA
•Decreased urine concentration (low specific gravity)
•Complex changes in cytokines, interferones, NK cells, white blood cell indicators, etc.
...
Symptomen van een dopaminestoornis
Symptomen van een dopaminestoornis
zit in pompoenzaden, amandelen, avocado's, bananen, haring, kaas, sesamzaad
Niet geschikt bij migraine, hoge bloeddruk of tegelijkertijd met antidepressiva
Extra:
Vit B = nodig voor aanmaak neurotransmitters
Vit C minimaal 2g
magnesium
Visolie EPA DHA 1 g
Goed supplement bij depressie algemeen:
Nutramin Emotio Balans, 1 x ochtend en 1 x avond
Niet bij zwangerschap of borstvoeding
- besluiteloosheid
- arm aan initiatieven
- depressie
- angst
- uitputting
- chronisch vermoeid, de hele dag kunnen slapen
- tremor
zit in pompoenzaden, amandelen, avocado's, bananen, haring, kaas, sesamzaad
Niet geschikt bij migraine, hoge bloeddruk of tegelijkertijd met antidepressiva
Extra:
Vit B = nodig voor aanmaak neurotransmitters
Vit C minimaal 2g
magnesium
Visolie EPA DHA 1 g
Goed supplement bij depressie algemeen:
Nutramin Emotio Balans, 1 x ochtend en 1 x avond
Niet bij zwangerschap of borstvoeding
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