vrijdag 2 maart 2012
woensdag 29 februari 2012
Survey: The statin study effect
https://www.statineffects.com/
Have you had any side effects from statin treatment?
If you would like to share your experience on statins with researchers at the University of California in San Diego ,
please email at statinstudy@ucsd.edu
and/or complete an online (paperless) survey at
http://www.statineffects.com
Have you had any side effects from statin treatment?
If you would like to share your experience on statins with researchers at the University of California in San Diego ,
please email at statinstudy@ucsd.edu
and/or complete an online (paperless) survey at
http://www.statineffects.com
zondag 26 februari 2012
The hidden cure for viral diseases?
http://www.ceri.com/BHTbook-StevenWmFowkes-100903.pdf
This online book offers a biologically sustainable solution to chronic viral disease (herpes, CMV, ...)
The treatment is about the drug BTH (butylated hydroxytoluene). It will cost you 10$ in bulk or 50$ in capsules. Show it to your doctor.
From what I read, it looks promising. Take your time or have someone take notes for you.
This online book offers a biologically sustainable solution to chronic viral disease (herpes, CMV, ...)
The treatment is about the drug BTH (butylated hydroxytoluene). It will cost you 10$ in bulk or 50$ in capsules. Show it to your doctor.
From what I read, it looks promising. Take your time or have someone take notes for you.
vrijdag 24 februari 2012
Duidelijkheid rond vitamine B12
Naar aanleiding van de ervaringen van een patient met vitamine B12, deel ik hier zijn vaststellingen.
Hij was ernstig beperkt gedurende vele jaren maar is nu bijna volledig hersteld. De moeite waard om het eens door te nemen. Veel mensen prikken of slikken immers vitamine B12.
http://aboutmecfs.org.violet.arvixe.com/Trt/B12Ineffectiveness.aspx
Zelf heb ik geëxperimenteerd met verscheidene injecteerbare producten omdat de vloeistof zo pijnlijk blijkt. Soms kon ik gedurende een dag mijn bed niet meer uit van de pijn. Andere mensen hebben hier ook last van, anderen dan weer niet.
Mogelijk ligt het antwoord in zijn (vertaalde) tekst hieronder.
Niet iedereen blijkt "vitamine B12" te kunnen omzetten. De oorzaken zijn divers:
Hij was ernstig beperkt gedurende vele jaren maar is nu bijna volledig hersteld. De moeite waard om het eens door te nemen. Veel mensen prikken of slikken immers vitamine B12.
Hier vindt u het origineel:
http://aboutmecfs.org.violet.arvixe.com/Trt/B12Ineffectiveness.aspx
Zelf heb ik geëxperimenteerd met verscheidene injecteerbare producten omdat de vloeistof zo pijnlijk blijkt. Soms kon ik gedurende een dag mijn bed niet meer uit van de pijn. Andere mensen hebben hier ook last van, anderen dan weer niet.
Mogelijk ligt het antwoord in zijn (vertaalde) tekst hieronder.
Niet iedereen blijkt "vitamine B12" te kunnen omzetten. De oorzaken zijn divers:
- Onmogelijkheid om vit B12 te absorberen vanwege pernicieuze anemie (genetisch of auto-immuun)
- Men kan vit B12 niet binden voor transport
- Er zijn genetische uitdagingen
- Verlaagde cobalamine niveaus in ruggemerg - hypothetische oorzaak, bevestigd probleem
- Afwezigheid van enzym om methylb12 in adenosylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om adenosylb12 in methylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om cyanocobalamin in methylb12 of adenosylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om hydroxycobalamin in methylb12 of adenosylb12 om te zetten - bevestigd, cobalamin letter ziekte
- Afwezigheid van enzym om glutathionylcobalamin in methylb12 of adenosylb12 om te zetten - hypothetisch, enige aanwijzingen voor probleem
- Te weinig enzymen om foliumzuur om te zetten in de bio-actieve vorm, methylfolaat - bevestigde oorzaak
- Actieve vorm: vit B12 in orale vorm wordt slechts voor 1% opgenomen omdat het door een klein celgaatje moet en omdat de binding verminderd kan zijn door genetische of aangeworven redenen.
- Inactieve vorm = cyanoB12 of hydroxyB12
- voordeel: waarden in bloed stijgen snel boven 300 pg/ml
- nadeel: geneest de schade niet aangericht door tekort aan vit B12
- nadeel: mogelijk blokkeren ze de B12 receptoren op de cellen waardoor de echte B12 niet meer kan werken
- nadeel: beide vormen kunnen maar voor 10 mcg/dag door een celgaatje en dus beperkt omgezet worden
- nadeel: ze doen het niveau van de ongebonden cobalamine niet stijgen (en net deze heb je nodig voor herstel)
- Enzymen: Sommige mensen missen de enzymen om cyanoB12 en hydroxyB12 om te zetten in de actieve vorm
- Verkeerd gebruik: Zuigtabletten blijken alleen te werken als je ze gedurende 45 min tot 2 uur onder de bovenlip of onder de tong laat smelten.
- Verkeerd merk: Slechts enkele merken blijken effectief werkzaam waaronder:
- Enzymatic Therapy 1 mg
- Jarrow Formulas 1 en 5 mg
- Country Life Dibenzodide 1 mg (adenosylB12)
- Solgar Metalfolin (methylfolaat) is beter dan eender welk foliumzuur
- Injecteerbaar product: MethylB12 wordt afgebroken tot hydroxyB12 maar geneest geen mensen met een tekort aan cobalamine. Dat vereist ongebonden methylB12 én adenosylB12. Mensen nemen vaak slechts 1 soort vitamine B12.
- Te lage dosering: mensen nemen te lage doseringen om efficient het ruggemerg te beïnvloeden
- Te weinig methylfolaat: dit is de werkzame vorm van foliumzuur
- Tekort aan essentiële co-factoren
- Suppleren met glutathion of precursors van glutathion: kunnen mogelijk een actieve vit B12 deficiëntie inleiden
Persoonlijk heb ik een vraagteken bij het punt dat het geen schade zou herstellen n.a.v. een tekort. Zelfs indien het geen schade herstelt, merkte ik het eerste jaar toch een mindering van de brainfog en meer stamina zo'n 24 uur na een inspuiting. Hoe meer tijd ik tussen de spuitjes liet, hoe pijnlijker ze telkens weer werden.
Voor degenen die er meer over willen lezen:
Het verhaal van Fred: http://aboutmecfs.org/Trt/B12Story.aspx
Een intro over B12: http://aboutmecfs.org/Trt/B12Intro.aspx
Het behandelingsplan van Fred: http://aboutmecfs.org/Trt/B12Treatment.aspx
http://www.iherb.com/Jarrow-Formulas-Methyl-B-12-5000-mcg-60-Lozenges/117?at=0
http://www.iherb.com/Country-Life-Gluten-Free-Active-B-12-Dibencozide-3000-mcg-60-Lozenges/1637?at=0
http://www.iherb.com/Solgar-Folate-Metafolin-Folic-Acid-800-mcg-100-Tablets/13961?at=0
http://www.iherb.com/Natural-Factors-Potassium-Citrate-99-mg-90-Tablets/2627?at=0
EXTRA LINK: http://www.diagned.nl/files_content/Diagned38-3.pdf
Een intro over B12: http://aboutmecfs.org/Trt/B12Intro.aspx
Het behandelingsplan van Fred: http://aboutmecfs.org/Trt/B12Treatment.aspx
http://www.iherb.com/Jarrow-Formulas-Methyl-B-12-5000-mcg-60-Lozenges/117?at=0
http://www.iherb.com/Country-Life-Gluten-Free-Active-B-12-Dibencozide-3000-mcg-60-Lozenges/1637?at=0
http://www.iherb.com/Solgar-Folate-Metafolin-Folic-Acid-800-mcg-100-Tablets/13961?at=0
http://www.iherb.com/Natural-Factors-Potassium-Citrate-99-mg-90-Tablets/2627?at=0
EXTRA LINK: http://www.diagned.nl/files_content/Diagned38-3.pdf
maandag 20 februari 2012
Statin Adverse Effects: Evidence for a Mitochondrial Mechanism
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2849981/?tool=pmcentrez
Statin Adverse Effects: A Review of the Literature and Evidence for a Mitochondrial Mechanism
In the case of statins, a potential basis for opposing effects occurring in muscle and in other organs can be identified. Evidence supports the proposition that antioxidant effects of statins underlie (or contribute to) many fundamental statin benefits – including benefits to flow and oxygen delivery46-48 and inflammation.49, 50 These effects may participate in improved walking distance in patients on statins, including benefits to muscle/walking in persons with and without peripheral artery disease.29 Yet a subset of people reproducibly exhibit increases in markers of oxidation on statins,51 and the occurrence of this increase has been tied to muscle pain on statins.52, 53
...
A range of cases have now been reported in which statin use has “uncovered” previously clinically silent or clinically tolerated conditions, ranging from McArdle disease159, 160 to myotonic dystrophy159 to acid maltase deficiency161 to possible Kennedy disease.159 Statins have also exacerbated known muscle conditions, such as myasthenia gravis.78 In the case of mitochondrial myopathies, the relative degree to which statins have unmasked vs induced disease may not always be clear.159, 162
...
Several widely used statins – atorvastatin, simvastatin, and lovastatin (and previously cerivastatin, now off the market) – are metabolized by the cytochrome P450 (CYP)3A4 pathway.318
...
While a medley of potential mechanisms may cause or contribute to statin AEs (and these merit more full review in another venue), mitochondrial mechanisms have been repeatedly implicated in muscle AEs. Mitochondrial defects predispose to problems on statins (as shown in the second to last entry of Table IV, ‘Genetic mutations associated with mitochondrial dysfunction’). Additionally, statins predispose to mitochondrial defects (Table V,22 31, 32, 112, 155, 158, 162, 397, 406-414) – in all users and, to a greater degree, in vulnerable individuals. Dose-dependent reductions in coenzyme Q1020-22 can reduce cell energy, promote oxidation,362, 415 promote apoptosis, and unmask silent mitochondrial defects.23-25, 362, 415-418 The mevalonate pathway, which statins inhibit, also produces heme-A, which has it own central involvement in mitochondrial electron transport.419
...
For instance, mitochondrial encephalomyopathy resulting from heritable coenzyme Q10 deficiency classically produces fatigue, muscle symptoms, and cognitive problems,440 although the cases referred for analysis are often relatively severe.429, 441 Gastrointestinal26 and neurological symptoms,432, 442 psychiatric symptoms,443-446 sleep problems,444, 447 glucose elevations,182 and a range of other symptoms reported on statins also arise in mitochondrial dysfunction.379, 448-457
...
Thus, in an analysis of data, presented in the Australian Adverse Drug Reaction Bulletin, it was noted that “Statin-associated peripheral neuropathy may persist for months or years after withdrawal of the statin… In two ADRAC (Adverse Drug Reactions Advisory Committee) cases of persistent peripheral neuropathy, motor and sensory conduction tests showed minimal recovery 4 and 12 months, respectively, after discontinuation of simvastatin, despite clinical improvement.”561
As reviewed here, AEs on statins may signal a mitochondrial vulnerability, which may alter or perhaps even reverse an otherwise favorable impact of statins on cell energetics. And AEs may signal occurrence of a net prooxidant rather than antioxidant effect of statins53 with possible unfavorable implications for a range of statins' proposed pleiotropic effects.892
Full report here: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2849981/?tool=pmcentrez
Statin Adverse Effects: A Review of the Literature and Evidence for a Mitochondrial Mechanism
In the case of statins, a potential basis for opposing effects occurring in muscle and in other organs can be identified. Evidence supports the proposition that antioxidant effects of statins underlie (or contribute to) many fundamental statin benefits – including benefits to flow and oxygen delivery46-48 and inflammation.49, 50 These effects may participate in improved walking distance in patients on statins, including benefits to muscle/walking in persons with and without peripheral artery disease.29 Yet a subset of people reproducibly exhibit increases in markers of oxidation on statins,51 and the occurrence of this increase has been tied to muscle pain on statins.52, 53
...
A range of cases have now been reported in which statin use has “uncovered” previously clinically silent or clinically tolerated conditions, ranging from McArdle disease159, 160 to myotonic dystrophy159 to acid maltase deficiency161 to possible Kennedy disease.159 Statins have also exacerbated known muscle conditions, such as myasthenia gravis.78 In the case of mitochondrial myopathies, the relative degree to which statins have unmasked vs induced disease may not always be clear.159, 162
...
Several widely used statins – atorvastatin, simvastatin, and lovastatin (and previously cerivastatin, now off the market) – are metabolized by the cytochrome P450 (CYP)3A4 pathway.318
...
While a medley of potential mechanisms may cause or contribute to statin AEs (and these merit more full review in another venue), mitochondrial mechanisms have been repeatedly implicated in muscle AEs. Mitochondrial defects predispose to problems on statins (as shown in the second to last entry of Table IV, ‘Genetic mutations associated with mitochondrial dysfunction’). Additionally, statins predispose to mitochondrial defects (Table V,22 31, 32, 112, 155, 158, 162, 397, 406-414) – in all users and, to a greater degree, in vulnerable individuals. Dose-dependent reductions in coenzyme Q1020-22 can reduce cell energy, promote oxidation,362, 415 promote apoptosis, and unmask silent mitochondrial defects.23-25, 362, 415-418 The mevalonate pathway, which statins inhibit, also produces heme-A, which has it own central involvement in mitochondrial electron transport.419
...
For instance, mitochondrial encephalomyopathy resulting from heritable coenzyme Q10 deficiency classically produces fatigue, muscle symptoms, and cognitive problems,440 although the cases referred for analysis are often relatively severe.429, 441 Gastrointestinal26 and neurological symptoms,432, 442 psychiatric symptoms,443-446 sleep problems,444, 447 glucose elevations,182 and a range of other symptoms reported on statins also arise in mitochondrial dysfunction.379, 448-457
...
Thus, in an analysis of data, presented in the Australian Adverse Drug Reaction Bulletin, it was noted that “Statin-associated peripheral neuropathy may persist for months or years after withdrawal of the statin… In two ADRAC (Adverse Drug Reactions Advisory Committee) cases of persistent peripheral neuropathy, motor and sensory conduction tests showed minimal recovery 4 and 12 months, respectively, after discontinuation of simvastatin, despite clinical improvement.”561
As reviewed here, AEs on statins may signal a mitochondrial vulnerability, which may alter or perhaps even reverse an otherwise favorable impact of statins on cell energetics. And AEs may signal occurrence of a net prooxidant rather than antioxidant effect of statins53 with possible unfavorable implications for a range of statins' proposed pleiotropic effects.892
Full report here: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2849981/?tool=pmcentrez
Dr. Klinghardt's Treatment of Lyme Disease
http://articles.mercola.com/sites/articles/archive/2009/08/04/Dr-Klinghardts-Treatment-of-Lyme-Disease.aspx
Extract:
Regular lab parameters affected by Lyme:
•Abnormal lipid profile (moderate cholesterol elevation with significant LDL elevation)
•Insulin resistance
•Borderline low white blood cells, normal SED rate and CRP
•Normal thyroid hormone tests but positive Barnes test and excellent response to giving T3
•Type 2 (high cortisol, low DHEA) or type 3 adrenal failure (low cortisol and DHEA)
•Low testosterone and DHEA
•Decreased urine concentration (low specific gravity)
•Complex changes in cytokines, interferones, NK cells, white blood cell indicators, etc.
...
Extract:
Regular lab parameters affected by Lyme:
•Abnormal lipid profile (moderate cholesterol elevation with significant LDL elevation)
•Insulin resistance
•Borderline low white blood cells, normal SED rate and CRP
•Normal thyroid hormone tests but positive Barnes test and excellent response to giving T3
•Type 2 (high cortisol, low DHEA) or type 3 adrenal failure (low cortisol and DHEA)
•Low testosterone and DHEA
•Decreased urine concentration (low specific gravity)
•Complex changes in cytokines, interferones, NK cells, white blood cell indicators, etc.
...
Symptomen van een dopaminestoornis
Symptomen van een dopaminestoornis
zit in pompoenzaden, amandelen, avocado's, bananen, haring, kaas, sesamzaad
Niet geschikt bij migraine, hoge bloeddruk of tegelijkertijd met antidepressiva
Extra:
Vit B = nodig voor aanmaak neurotransmitters
Vit C minimaal 2g
magnesium
Visolie EPA DHA 1 g
Goed supplement bij depressie algemeen:
Nutramin Emotio Balans, 1 x ochtend en 1 x avond
Niet bij zwangerschap of borstvoeding
- besluiteloosheid
- arm aan initiatieven
- depressie
- angst
- uitputting
- chronisch vermoeid, de hele dag kunnen slapen
- tremor
zit in pompoenzaden, amandelen, avocado's, bananen, haring, kaas, sesamzaad
Niet geschikt bij migraine, hoge bloeddruk of tegelijkertijd met antidepressiva
Extra:
Vit B = nodig voor aanmaak neurotransmitters
Vit C minimaal 2g
magnesium
Visolie EPA DHA 1 g
Goed supplement bij depressie algemeen:
Nutramin Emotio Balans, 1 x ochtend en 1 x avond
Niet bij zwangerschap of borstvoeding
zondag 19 februari 2012
Symptomen van een serotoninestoornis
Symptomen van een serotoninestoornis
Supplement: Tryptofaan
= essentieel aminozuur, zit in melk, vlees en chocolade
= even effectief als antidepressivum (SSRI)
verbetert membraanfluiditeit
Vooraleer je aan de slag gaat met SAMe, lees deze link eerst:
http://www.alternativementalhealth.com/articles/walshQZ.htm
A quick way to test for need for methylation therapy is to carry out a cautious trial of SAMe. Within a week or two you should have your answer. If she clearly is improving on the SAMs (which is frightfully expensive)..... you can get usually the same benefits (albeit more slowly) using methionine plus calcium, magnesium, and B-6. This should be side-effect free unless (a) the methylation is begun too abruptly or (b) the patient has a rare genetic enzyme disorder which disrupts the SAM cycle. We've found that direct methylation is usually more successful than tinkering with the SAM cycle. The primary way humans receive most of their methyl groups is from dietary methionine. It's often hard to improve on Mother Nature. (Jan 20, 2003)
SAMe is likely to cause great worsening of symptoms, including mania, if given to an OVER-methylated person. The incidence of overmethylation in our patient database of 1,500 bipolar cases is about 18%. Bipolar disorder is not a single condition, but a collection of very different biochemical disorders under the same umbrella diagnosis. SAMe works great for truly undermethylated patients, but all hell breaks out if given to someone who is overloaded (genetically) with methyl groups. The right way to do this is to (a) first determine the person's innate methylation tendency & then (b) act accordingly. (Jan 31, 2003)
Multiple food & chemical sensitivities are also associated with histapenia (low histamine, overmethylation), the largest of all SZ groups, amounting to about 48% of all cases. For this group, SZ symptoms often worsen if exposed to the offending substances, & nice improvements often occur if they are identified & avoided. However, the food sensitivities usually disappear after about 1 year of aggressive Folate/B-12/B-3 treatment, which is the primary route to a normal life for these patients.
...
Histamine assays for depression were introduced by Dr. Carl Pfeiffer of Princeton, NJ in the 1970' and 1980's. My clinic has found whole blood histamine to be very useful & has used this assay more than 30,000 times.
First of all, the analysis must be done for whole blood (not plasma, serum, etc), strictly adhering to the sampling protocol. We presently use LabCorp but in the past Quest also had proficiency for this assay.
The reference "normal" range for mental health is 40 to 70 ng/dL. Levels above 70 indicate undermethylation, whereas levels below 40 suggest overmethylation.
Undermethylated depressives thrive on l-methionine, calcium, magnesium, B-6, Zinc, and Vitamin C. In severe cases, up to 3,000 mg/day of methionine and 2,000 mg/day of Ca may be needed. However, we also like to routinely run a homocysteine test to assure the safety of the methylation protocol. This population is believed to result in low serotonin activity. This methylation therapy is quite slow in taking effect.... and often 6-8 weeks pass before progress is obvious.
Overmethylated (low-histamine) depressives thrive on folic acid, B-12, niacin (or niacinamide), B-6, Zinc, Manganese, DMAE, and Vitamins, C and E. In severe cases, up to 5,000 mcg/day of FA may be needed. Response is more rapid with this phenotype, with clear progress usually by week 4. This population is believed to have an innate tendency for elevated serotonin, dopamine, and norepinephrine levels.
This test can also help guide psychiatrists in selection of psychiatric medications. For example high histamine persons may do quite well on SSRI's, but low-histamine persons usually reactly very badly to SSRI's and are better candidates for benzodiazapines.
We like to augment the histamine blood test with an "absolute basophil" test offered by Direct Healthcare, Inc. The histamine assay can be affected by antihistamines and other medications with AH properties. The reference range for ABC's is 30-50.
- slaapproblemen
- spijsverteringsproblemen
- te weinig serotonine geeft obstipatie
- teveel serotonine geeft diarrhee
- angst met diepe triestheid
- depressie
- verhoogde pijngevoeligheid
- hoofdpijn
- niet lekker in je vel zitten
- uitputting
Supplement: Tryptofaan
= essentieel aminozuur, zit in melk, vlees en chocolade
- L-tryptofaan: lichaam kan het omzetten naar believen en wat het nodig heeft
- 5 hydroxitryptofaan: kan alleen omgezet worden in serotonine en geeft soms bijwerkingen
Supplement: SAMe butaandisulfonaat
= lichaamseigen stof = even effectief als antidepressivum (SSRI)
verbetert membraanfluiditeit
Vooraleer je aan de slag gaat met SAMe, lees deze link eerst:
http://www.alternativementalhealth.com/articles/walshQZ.htm
A quick way to test for need for methylation therapy is to carry out a cautious trial of SAMe. Within a week or two you should have your answer. If she clearly is improving on the SAMs (which is frightfully expensive)..... you can get usually the same benefits (albeit more slowly) using methionine plus calcium, magnesium, and B-6. This should be side-effect free unless (a) the methylation is begun too abruptly or (b) the patient has a rare genetic enzyme disorder which disrupts the SAM cycle. We've found that direct methylation is usually more successful than tinkering with the SAM cycle. The primary way humans receive most of their methyl groups is from dietary methionine. It's often hard to improve on Mother Nature. (Jan 20, 2003)
SAMe is likely to cause great worsening of symptoms, including mania, if given to an OVER-methylated person. The incidence of overmethylation in our patient database of 1,500 bipolar cases is about 18%. Bipolar disorder is not a single condition, but a collection of very different biochemical disorders under the same umbrella diagnosis. SAMe works great for truly undermethylated patients, but all hell breaks out if given to someone who is overloaded (genetically) with methyl groups. The right way to do this is to (a) first determine the person's innate methylation tendency & then (b) act accordingly. (Jan 31, 2003)
Multiple food & chemical sensitivities are also associated with histapenia (low histamine, overmethylation), the largest of all SZ groups, amounting to about 48% of all cases. For this group, SZ symptoms often worsen if exposed to the offending substances, & nice improvements often occur if they are identified & avoided. However, the food sensitivities usually disappear after about 1 year of aggressive Folate/B-12/B-3 treatment, which is the primary route to a normal life for these patients.
...
Histamine assays for depression were introduced by Dr. Carl Pfeiffer of Princeton, NJ in the 1970' and 1980's. My clinic has found whole blood histamine to be very useful & has used this assay more than 30,000 times.
First of all, the analysis must be done for whole blood (not plasma, serum, etc), strictly adhering to the sampling protocol. We presently use LabCorp but in the past Quest also had proficiency for this assay.
The reference "normal" range for mental health is 40 to 70 ng/dL. Levels above 70 indicate undermethylation, whereas levels below 40 suggest overmethylation.
Undermethylated depressives thrive on l-methionine, calcium, magnesium, B-6, Zinc, and Vitamin C. In severe cases, up to 3,000 mg/day of methionine and 2,000 mg/day of Ca may be needed. However, we also like to routinely run a homocysteine test to assure the safety of the methylation protocol. This population is believed to result in low serotonin activity. This methylation therapy is quite slow in taking effect.... and often 6-8 weeks pass before progress is obvious.
Overmethylated (low-histamine) depressives thrive on folic acid, B-12, niacin (or niacinamide), B-6, Zinc, Manganese, DMAE, and Vitamins, C and E. In severe cases, up to 5,000 mcg/day of FA may be needed. Response is more rapid with this phenotype, with clear progress usually by week 4. This population is believed to have an innate tendency for elevated serotonin, dopamine, and norepinephrine levels.
This test can also help guide psychiatrists in selection of psychiatric medications. For example high histamine persons may do quite well on SSRI's, but low-histamine persons usually reactly very badly to SSRI's and are better candidates for benzodiazapines.
We like to augment the histamine blood test with an "absolute basophil" test offered by Direct Healthcare, Inc. The histamine assay can be affected by antihistamines and other medications with AH properties. The reference range for ABC's is 30-50.
vrijdag 17 februari 2012
Herprogrammeer uw hond
http://www.gva.be/nieuws/wetenschap/aid1120069/hond-aan-de-rilatine.aspx
Honden zijn het volgende slachtoffer van de farma-industrie. Ze worden nu ook aan de Rilatine en antidepressiva gezet. De farma-industrie heeft reeds alle koeien, varkens en kippen volgepropt met antibiotica. Dat volstond blijkbaar niet.
Dierengedragstherapeut Dany Grosemans zegt in een artikel spijtige trend". "In mijn praktijk zie ik iets helemaal anders. Ik heb de indruk dat de farmaceutische firma's de dierenartsen het hoofd gek maken. Het is zelfs zo ver gekomen dat ik in een krant een advertentie zag verschijnen van een farmaceutisch bedrijf met de slogan "Herprogrammeer uw hond".
Tiens, dat zei die biopsychosociale arts in het CVS-centrum ook tegen mij tijdens mijn eerste gesprek: "U moet geherprogrammeerd worden."
Het zal wel toeval zijn zeker ...
Bron: http://www.gva.be/nieuws/wetenschap/aid1120069/hond-aan-de-rilatine.aspx
Honden zijn het volgende slachtoffer van de farma-industrie. Ze worden nu ook aan de Rilatine en antidepressiva gezet. De farma-industrie heeft reeds alle koeien, varkens en kippen volgepropt met antibiotica. Dat volstond blijkbaar niet.
Dierengedragstherapeut Dany Grosemans zegt in een artikel spijtige trend". "In mijn praktijk zie ik iets helemaal anders. Ik heb de indruk dat de farmaceutische firma's de dierenartsen het hoofd gek maken. Het is zelfs zo ver gekomen dat ik in een krant een advertentie zag verschijnen van een farmaceutisch bedrijf met de slogan "Herprogrammeer uw hond".
Tiens, dat zei die biopsychosociale arts in het CVS-centrum ook tegen mij tijdens mijn eerste gesprek: "U moet geherprogrammeerd worden."
Het zal wel toeval zijn zeker ...
Bron: http://www.gva.be/nieuws/wetenschap/aid1120069/hond-aan-de-rilatine.aspx
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