Ik had me aan de klap van de week verwacht na de lenteschoonmaak van dit weekend. Niet dus. De low dose naltrexone verkort duidelijk de recuperatie na inspanning op termijn!
Ik verzamel informatie voor mijn project. Zolang ik het liggend uitvoer, blijft het lichaam functioneren. Mijn dysautonomie hou ik onder controle door de dag grotendeels liggend door te brengen.
woensdag 27 april 2011
Dag 409 Oplosmiddel
Geen gekke dingen doen na gisteren. De pijn in de bovenbenen is sterk aanwezig alsof ze levend weggevreten worden door een oplosmiddel. We blijven in bed om de spieren te sparen. Geen griepachtige of zware dysautonomie symptomen ditmaal. Komt goed uit want ik heb leesplannen. Dat gaat niet met pudding in je hoofd.
Dag 408 Lenteschoonmaak
Stralend weer voor een lenteschoonmaak. Als dat lijf nu eens mee wilde werken, was die rommel in de schuur op een weekend de deur uit. Nu kan je er amper een voet binnenzetten. De conditie was de afgelopen week afschuwelijk maar sinds gisteren lijkt het spook in de nacht verdwenen. Ik breek me er het hoofd niet meer over. Het is eigen aan een virale aandoening, it comes and it goes. Dus het is even weg.
De schuur gaat er (deels) aan vandaag. Met trage bewegingen verplaats ik houten plankjes, tuingerief, dozen prullaria, ... tot hubby anderhalf uur later komt kijken en beslist mee te werken. Tegen dan sta ik verstijfd en in total slow motion aan te geven wat in de vuilbak moet en wat niet. Delegeren gaat me goed af. Zo gaat die schuur nog opgeruimd geraken.
De schuur gaat er (deels) aan vandaag. Met trage bewegingen verplaats ik houten plankjes, tuingerief, dozen prullaria, ... tot hubby anderhalf uur later komt kijken en beslist mee te werken. Tegen dan sta ik verstijfd en in total slow motion aan te geven wat in de vuilbak moet en wat niet. Delegeren gaat me goed af. Zo gaat die schuur nog opgeruimd geraken.
Dag 407 Moet er nog zand zijn?
Vandaag komt er een vriendje spelen. Het is prachtig weer en de zandbak wordt bovengehaald. Mijn oudste zoon weigert mee te gaan om zandzakjes te gaan halen in de winkel. Zucht, ik had het beloofd aan de kleintjes.
Met trillende armen til ik de loodzware zakjes in de winkelkar. Het karretje is haast niet te controleren op de parking. Ik ram net geen geparkeerde auto’s. Met een laatste krachtinspanning gaan ze in de kofferbak. Het duurt even voor ik weer achter het stuur kan om huiswaarts te keren. Gelukkig wil de zoon ze wel uit de auto halen bij terugkeer.
Met trillende armen til ik de loodzware zakjes in de winkelkar. Het karretje is haast niet te controleren op de parking. Ik ram net geen geparkeerde auto’s. Met een laatste krachtinspanning gaan ze in de kofferbak. Het duurt even voor ik weer achter het stuur kan om huiswaarts te keren. Gelukkig wil de zoon ze wel uit de auto halen bij terugkeer.
maandag 25 april 2011
Vaccine Researcher Indicted for Fraud
According to CNBC, “Paul Thorsen, the principal coordinator of multiple studies funded by the CDC used to deny a vaccine/autism link was indicted April 13th on thirteen counts of fraud and nine counts of money-laundering. The charges relate to funding for work he conducted for the CDC which claimed to disprove associations between the mercury-based vaccine preservative, thimerosal, and increased rates of autism.” This was first reported by the consumer group, SafeMinds.
I remember when these studies came out. They came out in rapid-fire succession just after previously-released studies began to show there was a link between autism and mercury-preserved vaccines. I was not surprised that the Thorsen articles came out so fast as Big Pharma had to do something to quiet the growing complaints about mercury in vaccines.
Do I think that mercury-containing vaccines cause autism? Before I answer that, let me state a few points. I believe there is no justification for ever injecting in any living being the third most toxic element known to mankind. As the wise man Forrest Gump said, “Stupid is as stupid does.” What do you expect will happen when you inject a known neurotoxin, in large amounts, to a small baby?
I have no doubt that mercury toxicity, in large part from vaccines, is probably one factor responsible for the rapid rise of autism. Even if mercury is not one of the underlying causes of autism, common sense as well as biochemical sense would dictate that mercury exposure is not a good idea. In fact, mercury toxicity is a common problem in many of the patients that I see. However, in our toxic world, there are many other items we are exposed to on a daily basis that can also disrupt the normal neurological development in a newborn such as lead, arsenic, nickel, cadmium and bromine.
What can you do? Number one; don’t let anyone inject mercury in your body. Most of the flu vaccines still have mercury in them; they should be avoided. Next, don’t let any dentist put a mercury filling in your mouth. Any dentist that still defends the use of mercury does not understand biochemistry and does not deserve your business. Next, eat a healthy diet free of pesticides and other chemicals that are harmful to your body. Finally, take the appropriate supplements to ensure that your detoxification pathways are optimally functioning.
Which supplements should you take? There are many items that aid the detoxification pathways including vitamins C and E, alpha lipoic acid, magnesium and glutathione.
Bron: http://drdavidbrownstein.blogspot.com/
According to CNBC, “Paul Thorsen, the principal coordinator of multiple studies funded by the CDC used to deny a vaccine/autism link was indicted April 13th on thirteen counts of fraud and nine counts of money-laundering. The charges relate to funding for work he conducted for the CDC which claimed to disprove associations between the mercury-based vaccine preservative, thimerosal, and increased rates of autism.” This was first reported by the consumer group, SafeMinds.
I remember when these studies came out. They came out in rapid-fire succession just after previously-released studies began to show there was a link between autism and mercury-preserved vaccines. I was not surprised that the Thorsen articles came out so fast as Big Pharma had to do something to quiet the growing complaints about mercury in vaccines.
Do I think that mercury-containing vaccines cause autism? Before I answer that, let me state a few points. I believe there is no justification for ever injecting in any living being the third most toxic element known to mankind. As the wise man Forrest Gump said, “Stupid is as stupid does.” What do you expect will happen when you inject a known neurotoxin, in large amounts, to a small baby?
I have no doubt that mercury toxicity, in large part from vaccines, is probably one factor responsible for the rapid rise of autism. Even if mercury is not one of the underlying causes of autism, common sense as well as biochemical sense would dictate that mercury exposure is not a good idea. In fact, mercury toxicity is a common problem in many of the patients that I see. However, in our toxic world, there are many other items we are exposed to on a daily basis that can also disrupt the normal neurological development in a newborn such as lead, arsenic, nickel, cadmium and bromine.
What can you do? Number one; don’t let anyone inject mercury in your body. Most of the flu vaccines still have mercury in them; they should be avoided. Next, don’t let any dentist put a mercury filling in your mouth. Any dentist that still defends the use of mercury does not understand biochemistry and does not deserve your business. Next, eat a healthy diet free of pesticides and other chemicals that are harmful to your body. Finally, take the appropriate supplements to ensure that your detoxification pathways are optimally functioning.
Which supplements should you take? There are many items that aid the detoxification pathways including vitamins C and E, alpha lipoic acid, magnesium and glutathione.
Bron: http://drdavidbrownstein.blogspot.com/
zondag 17 april 2011
Waarom vitamine D ontsteking remt
Vitamin D: the alternative hypothesis.
Autoimmun Rev. 2009 Jul;8(8):639-44. Epub 2009 Feb 12.
Albert PJ, Proal AD, Marshall TG.
Weill Cornell Medical College, New York, NY 10065, USA. paa2013@med.cornell.edu
Abstract
Early studies on vitamin D showed promise that various forms of the "vitamin" may be protective against chronic disease, yet systematic reviews and longer-term studies have failed to confirm these findings. A number of studies have suggested that patients with autoimmune diagnoses are deficient in 25-hydroxyvitamin D (25-D) and that consuming greater quantities of vitamin D, which further elevates 25 D levels, alleviates autoimmune disease symptoms.
Some years ago, molecular biology identified 25 D as a secosteroid. Secosteroids would typically be expected to depress inflammation, which is in line with the reports of symptomatic improvement. The simplistic first-order mass-action model used to guide the early vitamin studies is now giving way to a more complex description of action. When active, the Vitamin D nuclear receptor (VDR) affects transcription of at least 913 genes and impacts processes ranging from calcium metabolism to expression of key antimicrobial peptides. Additionally, recent research on the Human Microbiome shows that bacteria are far more pervasive than previously thought, increasing the possibility that autoimmune disease is bacterial in origin. Emerging molecular evidence suggests that symptomatic improvements among those administered vitamin D is the result of 25-D's ability to temper bacterial-induced inflammation by slowing VDR activity. While this results in short-term palliation, persistent pathogens that may influence disease progression, proliferate over the long-term.
Autoimmun Rev. 2009 Jul;8(8):639-44. Epub 2009 Feb 12.
Albert PJ, Proal AD, Marshall TG.
Weill Cornell Medical College, New York, NY 10065, USA. paa2013@med.cornell.edu
Abstract
Early studies on vitamin D showed promise that various forms of the "vitamin" may be protective against chronic disease, yet systematic reviews and longer-term studies have failed to confirm these findings. A number of studies have suggested that patients with autoimmune diagnoses are deficient in 25-hydroxyvitamin D (25-D) and that consuming greater quantities of vitamin D, which further elevates 25 D levels, alleviates autoimmune disease symptoms.
Some years ago, molecular biology identified 25 D as a secosteroid. Secosteroids would typically be expected to depress inflammation, which is in line with the reports of symptomatic improvement. The simplistic first-order mass-action model used to guide the early vitamin studies is now giving way to a more complex description of action. When active, the Vitamin D nuclear receptor (VDR) affects transcription of at least 913 genes and impacts processes ranging from calcium metabolism to expression of key antimicrobial peptides. Additionally, recent research on the Human Microbiome shows that bacteria are far more pervasive than previously thought, increasing the possibility that autoimmune disease is bacterial in origin. Emerging molecular evidence suggests that symptomatic improvements among those administered vitamin D is the result of 25-D's ability to temper bacterial-induced inflammation by slowing VDR activity. While this results in short-term palliation, persistent pathogens that may influence disease progression, proliferate over the long-term.
zondag 10 april 2011
Jodium in druppelvorm heeft ongekende toepassingsgebieden
Iodine is by far the best antibiotic, antiviral and antiseptic of all time - Dr. David Derry
Dr. Derry says that iodine is effective "for standard pathogens such as Staphylococcus, but also iodine has the broadest range of action, fewest side effects and no development of bacterial resistance." There is a world of difference between using an antibiotic – anti-life substance – and an antibiotic, antiviral and antifungal substance like iodine, which is life serving because it is a basic and most necessary nutritional substance.
Iodine kills single celled organisms by combining with the amino acids tyrosine or histidine when they are exposed to the extra-cellular environment. All single cells showing tyrosine on their outer cell membranes are killed instantly by a simple chemical reaction with iodine that denatures proteins. Nature and evolution have given us an important mechanism to control pathogenic life forms and we should use it and trust it to protect us in ways that antibiotics can't.
The way to combat antibiotic resistance is not bigger, better, stronger antibiotics but, rather, no antibiotics at all. Instead, other molecular weapons are available with the ability to disable bad germs without bothering good ones. Iodine is the ideal broad spectrum antibiotic that is not an antibiotic - it is not against life. Not against human life that is but you can hear the little pathogens screaming as high enough levels of iodine fan out through the system. Meaning all the viruses, bacteria, yeasts and molds that are threatening us are threatened with instant death when iodine is used orally to fight infection.
Bron: www.naturalnews.com/022800_antibiotic_antibiotics_infection.html
Dr. Derry says that iodine is effective "for standard pathogens such as Staphylococcus, but also iodine has the broadest range of action, fewest side effects and no development of bacterial resistance." There is a world of difference between using an antibiotic – anti-life substance – and an antibiotic, antiviral and antifungal substance like iodine, which is life serving because it is a basic and most necessary nutritional substance.
Iodine kills single celled organisms by combining with the amino acids tyrosine or histidine when they are exposed to the extra-cellular environment. All single cells showing tyrosine on their outer cell membranes are killed instantly by a simple chemical reaction with iodine that denatures proteins. Nature and evolution have given us an important mechanism to control pathogenic life forms and we should use it and trust it to protect us in ways that antibiotics can't.
The way to combat antibiotic resistance is not bigger, better, stronger antibiotics but, rather, no antibiotics at all. Instead, other molecular weapons are available with the ability to disable bad germs without bothering good ones. Iodine is the ideal broad spectrum antibiotic that is not an antibiotic - it is not against life. Not against human life that is but you can hear the little pathogens screaming as high enough levels of iodine fan out through the system. Meaning all the viruses, bacteria, yeasts and molds that are threatening us are threatened with instant death when iodine is used orally to fight infection.
Bron: www.naturalnews.com/022800_antibiotic_antibiotics_infection.html
Mutating viruses
Did you know that a nutritional deficiency can cause a virus to mutate to a more virulent form? That is the news from the United States Department of Agriculture (USDA) who are reporting that a human virus, normally harmless in laboratory mice, mutated into a heart-damaging pathogen when the animals were raised on a diet devoid of the essential element selenium. And, once mutated, the virus continued to damage hearts - even in mice that got ample selenium in their feed.
The importance of this is not limited to nutritionally-deprived populations, say researchers with the University of North Carolina and Agricultural Research Service of the government, who collaborated on the studies. In theory, one selenium-deficient person or animal could produce a new family of virus mutants that could cross species and spread worldwide, causing disease even in well nourished people.
The USDA is now officially on record that nutritional deficiencies cause viral mutations and they expect to find the same results with vitamin-E-deficient mice because both selenium and vitamin E are nutrients that serve as antioxidants in the body. This means that the government is recognizing that free radicals and oxidative stress affects the world of pathogens creating super bugs out of regular critters. They are even going as far as saying that this may help explain the many new strains of influenza virus arising in China, which has widespread selenium-deficient areas.
Bron: www.naturalnews.com/022800_antibiotic_antibiotics_infection.html#ixzz1J6Sn9u43
The importance of this is not limited to nutritionally-deprived populations, say researchers with the University of North Carolina and Agricultural Research Service of the government, who collaborated on the studies. In theory, one selenium-deficient person or animal could produce a new family of virus mutants that could cross species and spread worldwide, causing disease even in well nourished people.
The USDA is now officially on record that nutritional deficiencies cause viral mutations and they expect to find the same results with vitamin-E-deficient mice because both selenium and vitamin E are nutrients that serve as antioxidants in the body. This means that the government is recognizing that free radicals and oxidative stress affects the world of pathogens creating super bugs out of regular critters. They are even going as far as saying that this may help explain the many new strains of influenza virus arising in China, which has widespread selenium-deficient areas.
Bron: www.naturalnews.com/022800_antibiotic_antibiotics_infection.html#ixzz1J6Sn9u43
vrijdag 8 april 2011
Dysautonomie is een biomarker in ME
NIH workshop State of the Knowledge ME/CFS
April 07, 2011
Dr Rowe roept op om dysautonomie, POTS, orthostatische intoleratie als biomarker in ME te gebruiken.
http://www.youtube.com/watch?v=6KSPsUx4tFs
Hij begrijpt niet hoe psychiaters zoals Prins de schuld bij de patient blijven leggen. http://www.ncbi.nlm.nih.gov/pubmed/16443043
Of hoe psychiaters verantwoordelijk kunnen zijn voor de verspreiding van een epidemie ...
April 07, 2011
Dr Rowe roept op om dysautonomie, POTS, orthostatische intoleratie als biomarker in ME te gebruiken.
http://www.youtube.com/watch?v=6KSPsUx4tFs
Hij begrijpt niet hoe psychiaters zoals Prins de schuld bij de patient blijven leggen. http://www.ncbi.nlm.nih.gov/pubmed/16443043
Of hoe psychiaters verantwoordelijk kunnen zijn voor de verspreiding van een epidemie ...
dinsdag 5 april 2011
http://www.futuremedicine.com/doi/abs/10.2217/17460794.2.2.183
Virology of the post-polio syndrome
Andreina Baj, Salvatore Monaco, Gianluigi Zanusso, Elisa Dall’ora, Laura Bertolasi & Antonio Toniolo†† Author for correspondence
The three poliovirus serotypes (PVs) cause acute paralytic poliomyelitis. Decades after being hit by polio, survivors may develop a condition known as post-polio syndrome (PPS).
PPS is characterized by extreme fatigue, progressing muscular weakness and chronic pain. The pathogenesis is unclear and, thus, empirical therapies are employed.
PVs are known to be able to persist in infected host cells both in vitro and in vivo. The understanding of PV genomes has made it possible to set up sensitive and specific molecular tests capable of detecting minute amounts of virus in samples from PPS patients.
Current data indicate that complete PV genomes (or genomic fragments) remain present, decades after acute paralysis, in the CNS of these patients. Virus persistence is hypothesized to bring about chronic inflammation, immune-mediated injury and decreased expression of neurotrophic factors.
Establishing a pathogenetic link between PV persistence and PPS would be extremely relevant to the development of an etiologic therapy aimed at virus eradication.
Ik kijk ernaar uit! Het eerste artikel omtrent M.E. dat verscheen in 1956 maakte reeds vermelding van een mogelijke link met het polio-virus.
Virology of the post-polio syndrome
Andreina Baj, Salvatore Monaco, Gianluigi Zanusso, Elisa Dall’ora, Laura Bertolasi & Antonio Toniolo†† Author for correspondence
The three poliovirus serotypes (PVs) cause acute paralytic poliomyelitis. Decades after being hit by polio, survivors may develop a condition known as post-polio syndrome (PPS).
PPS is characterized by extreme fatigue, progressing muscular weakness and chronic pain. The pathogenesis is unclear and, thus, empirical therapies are employed.
PVs are known to be able to persist in infected host cells both in vitro and in vivo. The understanding of PV genomes has made it possible to set up sensitive and specific molecular tests capable of detecting minute amounts of virus in samples from PPS patients.
Current data indicate that complete PV genomes (or genomic fragments) remain present, decades after acute paralysis, in the CNS of these patients. Virus persistence is hypothesized to bring about chronic inflammation, immune-mediated injury and decreased expression of neurotrophic factors.
Establishing a pathogenetic link between PV persistence and PPS would be extremely relevant to the development of an etiologic therapy aimed at virus eradication.
Ik kijk ernaar uit! Het eerste artikel omtrent M.E. dat verscheen in 1956 maakte reeds vermelding van een mogelijke link met het polio-virus.
Abonneren op:
Posts (Atom)